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dc.contributor.authorRadulovic, Maja
dc.contributor.authorStenmark, Harald Alfred
dc.date.accessioned2021-09-27T11:50:42Z
dc.date.available2021-09-27T11:50:42Z
dc.date.created2021-02-05T15:58:51Z
dc.date.issued2020
dc.identifier.citationEMBO Journal. 2020, 39 (18), 1-3.en_US
dc.identifier.issn0261-4189
dc.identifier.urihttps://hdl.handle.net/11250/2783731
dc.description.abstractMutations in several genes encoding for lysosomal proteins are involved in Parkinson's disease (PD). In this issue, Herbst et al (2020) show that PD-related leucine-rich repeat kinase 2 (LRRK2) is activated in response to pathogen or membranolytic drug-induced damage of phagolysosomes and lysosomes in macrophages, and regulates endolysosomal homeostasis by controlling the balance between membrane repair and degradation.en_US
dc.language.isoengen_US
dc.publisherEMBO Pressen_US
dc.titleLRRK2 to the rescue of damaged endomembranesen_US
dc.typePeer revieweden_US
dc.typeJournal articleen_US
dc.description.versionacceptedVersionen_US
dc.rights.holderThis is the authors' accepted manuscript to an article published by EMBO Press.en_US
dc.source.pagenumber1-3en_US
dc.source.volume39en_US
dc.source.journalEMBO Journalen_US
dc.source.issue18en_US
dc.identifier.doi10.15252/embj.2020106162
dc.identifier.cristin1887181
dc.relation.projectNorges forskningsråd: 223255en_US
cristin.ispublishedtrue
cristin.fulltextoriginal
cristin.fulltextpostprint
cristin.qualitycode2


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