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dc.contributor.authorSkovereng, Knut
dc.contributor.authorEttema, Gertjan
dc.contributor.authorVan Beekvelt, Mireille
dc.identifier.citationPLoS ONE. 2017, 12:e0169573 (1), 1-13.nb_NO
dc.description.abstractThe purpose of the present study was to investigate the effect of cadence on joint specific power and cycling kinematics in the ankle joint in addition to muscle oxygenation and muscle VO2 in the gastrocnemius and tibialis anterior. Thirteen cyclists cycled at a cadence of 60, 70, 80, 90, 100 and 110 rpm at a constant external work rate of 160.1 ± 21.3 W. Increasing cadence led to a decrease in ankle power in the dorsal flexion phase and to an increase in ankle joint angular velocity above 80 rpm. In addition, increasing cadence increased deoxygenation and desaturation for both the gastrocnemius and tibialis anterior muscles. Muscle VO2 increased following increased cadence but only in the tibialis anterior and only at cadences above 80 rpm, thus coinciding with the increase in ankle joint angular velocity. There was no effect of cadence in the gastrocnemius. This study demonstrates that high cadences lead to increased mVO2 in the TA muscles that cannot be explained by power in the dorsal flexion phase.nb_NO
dc.publisherPublic Library of Sciencenb_NO
dc.rightsNavngivelse 4.0 Internasjonal*
dc.titleThe effect of cadence on shank muscle oxygen consumption and deoxygenation in relation to joint specific power and cycling kinematicsnb_NO
dc.typeJournal articlenb_NO
dc.typePeer reviewednb_NO
dc.source.journalPLoS ONEnb_NO
dc.description.localcode© 2017 Skovereng et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.nb_NO
cristin.unitnameInstitutt for nevromedisin

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Navngivelse 4.0 Internasjonal
Except where otherwise noted, this item's license is described as Navngivelse 4.0 Internasjonal