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Decreased adiponectin/leptin ratio relates to insulin resistance in adults with obesity

dc.contributor.authorCastela, Inês
dc.contributor.authorMorais, Juliana
dc.contributor.authorBarreiros-Mota, Inês
dc.contributor.authorSilvestre, Marta P.
dc.contributor.authorMarques, Cláudia
dc.contributor.authorRodrigues, Catarina
dc.contributor.authorIsmael, Shámila
dc.contributor.authorAraújo, João R
dc.contributor.authorÂngelo-Dias, Miguel
dc.contributor.authorMartins, Catarina
dc.contributor.authorBorrego, Luís Miguel
dc.contributor.authorMonteiro, Rosário
dc.contributor.authordos Santos Ribeiro F. Coutinho, Silvia
dc.contributor.authorCalhau, Conceição
dc.contributor.authorAlho Letra Martins, Catia Patricia
dc.contributor.authorFaria, Ana
dc.contributor.authorPestana, Diogo
dc.contributor.authorTeixeira, Diana
dc.date.accessioned2024-03-12T07:23:18Z
dc.date.available2024-03-12T07:23:18Z
dc.date.created2023-02-28T11:25:45Z
dc.date.issued2023
dc.identifier.citationAmerican Journal of Physiology. Endocrinology and Metabolism. 2023, 324 (2), E115-E119.en_US
dc.identifier.issn0193-1849
dc.identifier.urihttps://hdl.handle.net/11250/3121867
dc.description.abstractAdiponectin/leptin ratio, triacylglycerols, and waist-to-hip ratio explained almost half of HOMA-IR variance in the context of obesity. This study provides evidence to support adipose tissue dysfunction as a central feature of the pathophysiology of obesity and insulin resistance. Early identification of individuals at higher risk of developing metabolic complications through adipose tissue dysfunction assessment and the staging of obesity and its transient phenotypes can contribute to improve therapeutic decision-making.en_US
dc.language.isoengen_US
dc.publisherAmerican Physiological Societyen_US
dc.titleDecreased adiponectin/leptin ratio relates to insulin resistance in adults with obesityen_US
dc.title.alternativeDecreased adiponectin/leptin ratio relates to insulin resistance in adults with obesityen_US
dc.typeJournal articleen_US
dc.typePeer revieweden_US
dc.description.versionacceptedVersionen_US
dc.source.pagenumberE115-E119en_US
dc.source.volume324en_US
dc.source.journalAmerican Journal of Physiology. Endocrinology and Metabolismen_US
dc.source.issue2en_US
dc.identifier.doi10.1152/ajpendo.00273.2022
dc.identifier.cristin2130003
cristin.ispublishedtrue
cristin.fulltextpostprint
cristin.qualitycode1


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