dc.contributor.author | Shim, Jung Min | |
dc.contributor.author | Kim, Jinhee | |
dc.contributor.author | Tenson, Tanel | |
dc.contributor.author | Min, Ji-Young | |
dc.contributor.author | Kainov, Denis | |
dc.date.accessioned | 2018-06-28T11:07:35Z | |
dc.date.available | 2018-06-28T11:07:35Z | |
dc.date.created | 2017-11-03T13:18:40Z | |
dc.date.issued | 2017 | |
dc.identifier.citation | Viruses. 2017, 9(8). | nb_NO |
dc.identifier.issn | 1999-4915 | |
dc.identifier.uri | http://hdl.handle.net/11250/2503562 | |
dc.description.abstract | Human influenza A viruses (IAVs) cause global pandemics and epidemics, which remain serious threats to public health because of the shortage of effective means of control. To combat the surge of viral outbreaks, new treatments are urgently needed. Developing new virus control modalities requires better understanding of virus-host interactions. Here, we describe how IAV infection triggers cellular apoptosis and how this process can be exploited towards the development of new therapeutics, which might be more effective than the currently available anti-influenza drugs. | nb_NO |
dc.language.iso | eng | nb_NO |
dc.publisher | MDPI AG | nb_NO |
dc.rights | Navngivelse 4.0 Internasjonal | * |
dc.rights.uri | http://creativecommons.org/licenses/by/4.0/deed.no | * |
dc.title | Influenza virus infection, interferon response, viral counter-response, and apoptosis | nb_NO |
dc.type | Journal article | nb_NO |
dc.type | Peer reviewed | nb_NO |
dc.description.version | publishedVersion | nb_NO |
dc.source.volume | 9 | nb_NO |
dc.source.journal | Viruses | nb_NO |
dc.source.issue | 8 | nb_NO |
dc.identifier.doi | 10.3390/v9080223 | |
dc.identifier.cristin | 1510750 | |
dc.description.localcode | © 2017 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/). | nb_NO |
cristin.unitcode | 194,65,15,0 | |
cristin.unitname | Institutt for klinisk og molekylær medisin | |
cristin.ispublished | true | |
cristin.fulltext | original | |
cristin.qualitycode | 1 | |